Herpes simplex - Èpès Senp
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Epes senp sou lèv la.
Èpès Senp (Herpes simplex) — Enfeksyon èpès senp sou dwèt yo pi souvan rive lakòz timoun piti pase granmoun.
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ReferencesEnfeksyon HSV-1 a pwogrese atravè enfeksyon prensipal sou selil epitelyal, ki swiv pa yon peryòd latans, sitou nan nè, epi li ka reyaktive. HSV-1 souvan lakòz premye epizòd eripsyon vesikulè, sitou sou bouch la ak sou mukoza jenital. Manifestasyon li yo varye soti nan èpès orolabial rive nan plizyè lòt kondisyon tankou folikulit èpès, enfeksyon po, patisipasyon okulè, epi ka grav tankou ansefalit èpès. Terapi antiviral ede jere enfeksyon HSV.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) se yon enfeksyon ki gaye toupatou, ki afekte anviwon 22 % adilt ki gen laj 12 an oswa plis, ki totalize 45 milyon adilt Ozetazini. Pandan ke HSV-1 anjeneral lakòz maleng nan bouch, li ka mennen tou nan blesi jenital. Sepandan, lè pasyan yo gen blesi jenital, HSV-2 se nòmalman enkyetid prensipal la. Sentòm yo souvan vag, tankou demanjezon jenital ak iritasyon, ki ka retade dyagnostik ak tretman. Reta sa a ka lakòz plis transmisyon bay moun ki pa enfekte.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) souvan lakòz enfeksyon tankou epes jenital ak maleng la fwa nan adolesan ak granmoun. Lè HSV enfekte yon tibebe pandan premye 4 a 6 semèn lavi li, li ka lakoz yon maladi grav ak konsekans grav. Byen vit dyagnostike enfeksyon HSV neonatal la enpòtan anpil pou anpeche maladi a vin pi mal, epi pou evite pwoblèm neurologik (menm lanmò).
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Enfeksyon Herpes simplex se trè komen epi li ka pase soti nan fanm ansent bay tibebe yo. Viris sa a ka lakòz pwoblèm sante grav oswa menm lanmò nan tibebe ki fèk fèt. Pandan ke li ra pandan gwosès li menm, li souvan rive pandan akouchman. Risk la pi wo si manman an vin enfekte nan dènye etap gwosès la. Sepandan, risk sa a ka redwi lè w itilize dwòg antiviral oswa chwazi yon seksyon C nan sèten sitiyasyon.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) kalite 1 ak 2 enfekte anpil moun atravè lemond. Anjeneral, viris la rete trankil nan selil nève apre li fin enfekte po a, men li ka reaktive pita, sa ki lakòz maleng la fwa. Pafwa, li mennen nan pwoblèm grav tankou enfeksyon nan je, enflamasyon sèvo, oswa konplikasyon danjere pou tibebe ki fenk fèt ak moun ki gen sistèm iminitè fèb. Pandan ke medikaman ki disponib yo ede kontwole enfeksyon, risk pou rezistans dwòg ak efè segondè rete yon enkyetid. Nou bezwen nouvo medikaman pou vize viris la pi byen.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Gen de kalite viris èpès senp (Herpes simplex), tip 1 (HSV-1) ak tip 2 (HSV-2). HSV-1 pi souvan lakòz enfeksyon nan bouch la, pandan ke HSV-2 pi souvan lakòz enfeksyon jenital. Yo transmèt pa kontak dirèk ak moun ki enfekte a. Èpès jenital klase kòm yon enfeksyon seksyèlman transmisib. Li ka pase bay tibebe pandan akouchman. Apre enfeksyon, viris yo vwayaje atravè nè sansoryèl pou rive nan selil nè, kote yo rete pou tout lavi. Kòz repete yo ka enkli: fonksyon iminitè ki febli, estrès, ak ekspoze a solèy.
Nan pifò ka, medikaman antiviral yo itilize sèlman lè sentòm yo grav. Yo ka preskri medikaman antiviral chak jou pou moun ki soufri enfeksyon souvan. Pa gen vaksen ki disponib, epi vaksen bardo a pa anpeche èpès senp. Tretman ak medikaman antiviral tankou acyclovir oswa valacyclovir ka diminye gravite sentòm yo.
Pousantaj moun ki enfekte atravè lemond ak HSV-1 oswa HSV-2 se ant 60 % ak 95 % nan granmoun. HSV-1 anjeneral enfekte pandan anfans. Yo estime ke 536 milyon moun atravè lemond (16 % popilasyon an) te enfekte ak HSV-2 depi 2003, ak pi gwo pousantaj lan jwenn pami fanm. Pifò moun ki gen HSV-2 pa reyalize ke yo enfekte.
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Evite nenpòt kontak fizik, tankou bo yon timoun, pandan ti anpoul yo prezan, paske kontak ka gaye enfeksyon an bay lòt moun. Ou ta dwe repoze epi evite bwè alkòl.
Acyclovir cream